*PLEASE HELP GET THIS INFORMATION OUT TO AS MANY PEOPLE AS POSSIBLE*
* Coming COVID Catastrophe
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*Open Letter to the WHO: Immediately Halt All Covid-19 Mass Vaccinations*
*[Geert Vanden Bossche, DMV, PhD, independent virologist and vaccine
expert, formerly employed at GAVI and The Bill & Melinda Gates Foundation]*
To all authorities, scientists and experts around the world, to whom this
concerns: the entire world population.
I am all but an antivaxxer. As a scientist I do not usually appeal to any
platform of this kind to make a stand on vaccine-related topics. As a
dedicated virologist and vaccine expert I only make an exception when
health authorities allow vaccines to be administered in ways that threaten
public health, most certainly when scientific evidence is being ignored.
The present extremely critical situation forces me to spread this emergency
call. As the unprecedented extent of human intervention in the
Covid-19pandemic is now at risk of resulting in a global catastrophe
without equal, this call cannot sound loudly and strongly enough.
As stated, I am not against vaccination. On the contrary, I can assure you
that each of the current vaccines have been designed, developed and
manufactured by brilliant and competent scientists. However, this type of
prophylactic vaccines are completely inappropriate, and even highly
dangerous, when used in mass vaccination campaigns during a viral pandemic.
Vaccinologists, scientists and clinicians are blinded by the positive
short-term effects in individual patents, but don’t seem to bother about
the disastrous consequences for global health. Unless I am scientifically
proven wrong, it is difficult to understand how current human interventions
will prevent circulating variants from turning into a wild monster.
Racing against the clock, I am completing my scientific manuscript, the
publication of which is, unfortunately, likely to come too late given the
ever increasing threat from rapidly spreading, highly infectious variants.
This is why I decided to already post a summary of my findings as well as
my keynote speech at the recent Vaccine Summit in Ohio on LinkedIn.
Last Monday, I provided international health organizations, including the
WHO, with my analysis of the current pandemic as based on scientifically
informed insights in the immune biology of Covid-19. Given the level of
emergency, I urged them to consider my concerns and to initiate a debate on
the detrimental consequences of further ‘viral immune escape’.
For those who are no experts in this field, I am attaching below a more
accessible and comprehensible version of the science behind this insidious
phenomenon.
While there is no time to spare, I have not received any feedback thus far.
Experts and politicians have remained silent while obviously still eager to
talk about relaxing infection prevention rules and ‘springtime freedom’. My
statements are based on nothing else but science. They shall only be
contradicted by science.
While one can barely make any incorrect scientific statements without being
criticized by peers, it seems like the elite of scientists who are
currently advising our world leaders prefer to stay silent. Sufficient
scientific evidence has been brought to the table.
Unfortunately, it remains untouched by those who have the power to act. How
long can one ignore the problem when there is at present massive evidence
that viral immune escape is now threatening humanity? We can hardly say we
didn’t know – or were not warned.
In this agonizing letter I put all of my reputation and credibility at
stake. I expect from you, guardians of mankind, at least the same. It is of
utmost urgency. Do open the debate. By all means: turn the tide!
Why mass vaccination amidst a pandemic creates an irrepressible monster
THE key question is: why does nobody seem to bother about viral immune
escape? Let me try to explain this by means of a more easily understood
phenomenon: Antimicrobial resistance. One can easily extrapolate this
scourge to resistance to our self-made ‘antiviral antibiotics’. Indeed,
antibodies (Abs) produced by our own immune system can be considered
self-made antiviral antibiotics, regardless of whether they are part of our
innate immune system (so-called ‘natural’ Abs’) or elicited in response to
specific pathogens (resulting in so-called ‘acquired’ Abs).
Natural Abs are not germ-specific whereas acquired Abs are specifically
directed at the invading pathogen. At birth, our innate immune system is
‘unexperienced’ but well-established. It protects us from a multitude of
pathogens, thereby preventing these pathogens from causing disease.
As the innate immune system cannot remember the pathogens it encountered
(innate immunity has no so-called ‘immunological memory’), we can only
continue to rely on it provided we keep it ‘trained’ well enough.
Training is achieved by regular exposure to a myriad of environmental
agents, including pathogens. However, as we age, we will increasingly face
situations where our innate immunity (often called ‘the first line of
immune defense’) is not strong enough to halt the pathogen at the portal of
entry (mostly mucosal barriers like respiratory or intestinal epithelia).
When this happens, the immune system has to rely on more specialized
effectors of our immune system (i.e., antigen-specific Abs and T cells) to
fight the pathogen. So, as we grow up, we increasingly mount
pathogen-specific immunity, including highly specific Abs. As those have
stronger affinity for the pathogen (e.g., virus) and can reach high
concentrations, they can quite easily outcompete our natural Abs for
binding to the pathogen/virus.
It is precisely this type of highly specific, high affinity Abs that
current Covid-19 vaccines are inducing. Of course, the noble purpose of
these Abs is to protect us against Covid-19. So, why then should there be a
major concern using these vaccines to fight Covid-19?
Well, similar to the rules applying to classical antimicrobial antibiotics,
it is paramount that our self-made ‘antiviral antibiotics’ are made
available in sufficient concentration and are tailored at the specific
features of our enemy.
This is why in case of bacterial disease it is critical to not only chose
the right type of antibiotic (based on the results from an antibiogram) but
to also take the antibiotic for long enough (according to the prescription).
Failure to comply with these requirements is at risk of granting microbes a
chance to survive and hence, may cause the disease to fare up. A very
similar mechanism may also apply to viruses, especially to viruses that can
easily and rapidly mutate (which is, for example, the case with
Coronaviruses); when the pressure exerted by the army’s (read:
population’s) immune defense starts to threaten viral replication and
transmission, the virus will take on another coat so that it can no longer
be easily recognized and, therefore, attacked by the host immune system.
The virus is now able to escape immunity (so-called: ‘immune escape’).
However, the virus can only rely on this strategy provided it still has
room enough to replicate. Viruses, in contrast to the majority of bacteria,
must rely on living host cells to replicate. This is why the occurrence of
‘escape mutants’ isn’t too worrisome as long as the likelihood for these
variants to rapidly find another host is quite remote. However, that’s not
particularly the case during a viral pandemic!
During a pandemic, the virus is spreading all over the globe with many
subjects shedding and transmitting the virus (even including asymptomatic
‘carriers’). The higher the viral load, the higher the likelihood for the
virus to bump into subjects who haven’t been infected yet or who were
infected but didn’t develop symptoms. Unless they are sufficiently
protected by their innate immune defense (through natural Abs), they will
catch Covid-19 disease as they cannot rely on other, i.e., acquired Abs.
It has been extensively reported, indeed, that the increase in S
(spike)-specific Abs in asymptomatically infected people is rather limited
and only short-lived. Furthermore, these Abs have not achieved full
maturity.
The combination of viral infection on a background of suboptimal Ab
maturity and concentration enables the virus to select mutations allowing
it to escape the immune pressure. The selection of those mutations
preferably occurs in the S protein as this is the viral protein that is
responsible for viral infectiousness.
As the selected mutations endow the virus with increased infectious
capacity, it now becomes much easier for the virus to cause severe disease
in infected subjects. The more people develop symptomatic disease, the
better the virus can secure its propagation and perpetuation (people who
get severe disease will shed more virus and for a longer period of time
than asymptomatically infected subjects do).
Unfortunately, enough, the short-lived rise in S-specific Abs does,
however, surface to bypass people’s innate/natural Ab. Those are put out of
business as their affinity for S is lower than the affinity of S-specific
Abs. This is to say that with an increasing rate of infection in the
population, the number of subjects who get infected while experiencing a
momentary increase in S-specific Abs will steadily increase.
Consequently, the number of subjects who get infected while experiencing a
momentary decrease in their innate immunity will increase. As a result, a
steadily increasing number of subjects will become more susceptible to
getting severe disease instead of showing only mild symptoms (i.e., limited
to the upper respiratory tract) or no symptoms at all.
During a pandemic, especially youngsters will be affected by this evolution
as their natural Abs are not yet largely suppressed by a panoply of
‘acquired’, antigen-specific Abs. Natural Abs, and natural immunity in
general, play a critical role in protecting us from pathogens as they
constitute our first line of immune defense. In contrast to acquired
immunity, innate immune responses protect against a large spectrum of
pathogens (so don’t compromise or sacrifice your innate immune defense!).
Because natural Abs and innate immune cells recognize a diversified
spectrum of foreign (i.e., non-self) agents (only some of which have
pathogenic potential), it’s important, indeed, to keep it sufficiently
exposed to environmental challenges.
By keeping the innate immune system (which, unfortunately, has no memory!)
TRAINED, we can much more easily resist germs which have real pathogenic
potential. It has, for example, been reported and scientifically proven
that exposure to other, quite harmless Coronaviruses causing a ‘common cold
’ can provide protection, although short-lived, against Covid-19 and its
loyal henchmen (i.e., the more infectious variants).
Suppression of innate immunity, especially in the younger age groups, can,
therefore, become very problematic. There can be no doubt that lack of
exposure due to stringent containment measures implemented as of the
beginning of the pandemic has not been beneficial to keeping people’s
innate immune system well trained.
As if this was not already heavily compromising innate immune defense in
this population segment, there comes yet another force into play that will
dramatically enhance morbidity and mortality rates in the younger age
groups: MASS VACCINATION of the ELDERLY.
The more extensively the later age group will be vaccinated and hence,
protected, the more the virus is forced to continue causing
disease in younger age groups.
This is only going to be possible provided it escapes to the S-specific Abs
that are momentarily raised in previously asymptomatically infected
subjects. If the virus manages to do so, it can benefit from the
(momentarily) suppressed innate immunity, thereby causing disease in an
increasing number of these subjects and ensuring its own propagation.
Selecting targeted mutations in the S protein is, therefore, the way to go
in order for the virus to enhance its infectiousness in candidates that are
prone to getting the disease because of a transient weakness of their
innate immune defense.
But in the meantime, we’re also facing a huge problem in vaccinated people
as they’re now more and more confronted with infectious variants displaying
a type of S protein that is increasingly different from the S edition
comprised with the vaccine (the later edition originates from the original,
much less infectious strain at the beginning of the pandemic).
The more variants become infectious (i.e., as a result of blocking access
of the virus to the vaccinated segment of the population), the less
vaccinal Abs will protect. Already now, lack of protection is leading to
viral shedding and transmission in vaccine recipients who are exposed to
these more infectious strains (which, by the way, increasingly dominate the
field).
This is how we are currently turning vaccines into asymptomatic carriers
shedding infectious variants.
At some point, in a likely very near future, it’s going to become more
profitable (in term of ‘return on selection investment’) for the virus to
just add another few mutations (maybe just one or two) to the S protein of
viral variants (already endowed with multiple mutations enhancing
infectiousness) in an attempt to further strengthen its binding to the
receptor (ACE-2) expressed on the surface of permissive epithelial cells.
This will now allow the new variant to outcompete vaccinal Abs for binding
to the ACE receptor. This is to say that at this stage, it would only take
very few additional targeted mutations within the viral receptor-binding
domain to fully resist S-specific ant-Covid-19 Abs, regardless whether the
later are elicited by the vaccine or by natural infection.
At that stage, the virus will, indeed, have managed to gain access to a
huge reservoir of subjects who have now become highly susceptible to
disease as their S-specific Abs have now become useless in terms of
protection but still manage to provide for long-lived suppression of their
innate immunity (i.e., natural infection, and especially vaccination,
elicit relatively long-lived specific Ab titers). The susceptible reservoir
comprises both, vaccinated people and those who’re left with sufficient
S-specific Abs due to previous Covid-19 disease).
So, MISSION ACCOMPLISHED for Covid-19 but a DISASTROUS SITUATION for all
vaccinated subjects and Covid-19 seropositive people as they’ve now lost
both, their acquired and innate immune defense against Covid-19 (while
highly infectious strains are circulating!).
That’s ‘one small step for the virus, one giant catastrophe for mankind’,
which is to say that we’ll have whipped up the virus in the younger
population up to a level that it now takes little effort for Covid-19 to
transform into a highly infectious virus that completely ignores both the
innate arm of our immune system as well as the adaptive/acquired one
(regardless of whether the acquired Abs resulted from vaccination or
natural infection).
The effort for the virus is now becoming even more negligible given that
many vaccine recipients are now exposed to highly infectious viral variants
while having received only a single shot of the vaccine.
Hence, they are endowed with Abs that have not yet acquired optimal
functionality. There is no need to explain that this is just going to
further enhance immune escape. Basically, we’ll very soon be confronted
with a super-infectious virus that completely resists our most precious
defense mechanism: The human immune system.
From all of the above, it’s becoming increasingly difficult to imagine how
the consequences of the extensive and erroneous human intervention in this
pandemic are not going to wipe out large parts of our human population.
One could only think of very few other strategies to achieve the same level
of efficiency in turning a relatively harmless virus into a bioweapon of
mass destruction.
It’s certainly also worth mentioning that mutations in the S protein (i.e.,
exactly the same protein that is subject to selection of escape mutations)
are known to enable Coronaviruses to cross species barriers.
This is to say that the risk that vaccine-mediated immune escape could
allow the virus to jump to other animal species, especially industrial
livestock (e.g., pig and poultry farms), is not negligible.
These species are already known to host several different Coronaviruses and
are usually housed in farms with high stocking density.
Similar to the situation with influenza virus, these species could than
serve as an additional reservoir for SARS-COVID-2 virus.
As pathogens have co-evolved with the host immune system, natural pandemics
of acute self-limiting viral infections have been shaped such as to take a
toll on human lives that is not higher than strictly required.
Due to human intervention, the course of this pandemic has been thoroughly
disturbed as of the very beginning. Widespread and stringent infection
prevention measures combined with mass vaccination campaigns using
inadequate vaccines will undoubtedly lead to a situation where the pandemic
is getting increasingly ‘out of control’.
Paradoxically, the only intervention that could offer a perspective to end
this pandemic (other than to let it run its disastrous course) is
…VACCINATION. Of course, the type of vaccines to be used would be
completely different of conventional vaccines in that they’re not inducing
the usual suspects, i.e., B and T cells, but NK cells.
There is, indeed, compelling scientific evidence that these cells play a
key role in facilitating complete elimination of Covid-19 at an early stage
of infection in asymptomatically infected subjects.
NK cells are part of the cellular arm of our innate immune system and,
alike natural Abs, they are capable of recognizing and attacking a broad
and diversified spectrum of pathogenic agents.
There is a sound scientific rationale to assume that it is possible to
‘prime’ NK cells in ways for them to recognize and kill Coronaviruses at
large (include all their variants) at an early stage of infection. NK cells
have increasingly been described to be endowed with the capacity to acquire
immunological memory.
By educating these cells in ways that enable them to durably recognize and
target Coronavirus-infected cells, our immune system could be perfectly
armed for a targeted attack to the universe of Coronaviruses prior to
exposure.
As NK cell-based immune defense provides sterilizing immunity and allows
for broad-spectrum and fast protection, it is reasonable to assume that
harnessing our innate immune cells is going to be the only type of human
intervention left to halt the dangerous spread of highly infectious
Covid-19 variants.
If we, human beings, are committed to perpetuating our species, we have no
choice left but to eradicate these highly infectious viral variants. This
will, indeed, require large vaccination campaigns. However, NK cell-based
vaccines will primarily enable our natural immunity to be better prepared
(memory!) and to induce herd immunity (which is exactly the opposite of
what current Covid-19 vaccines do as those increasingly turn vaccine
recipients into asymptomatic carriers who are shedding virus).
So, there is not one second left for gears to be switched and to replace
the current killer vaccines by life-saving vaccines.
I am appealing to the WHO and all stakeholders involved, no matter their
conviction, to immediately declare such action as THE SINGLE MOST IMPORTANT
PUBLIC HEALTH EMERGENCY OF INTERNATIONAL CONCERN.
*Running Time: 64 mins*
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